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After activation, the Wnt/-catenin pathway causes -catenin to become stable and move to the nucleus, where it uses T-cell factor/lymphoid enhancer-binding factor (TCF/LEF) transcription factors to activate and control downstream target genes, ultimately increasing the expression of genes related to cell migration, differentiation, proliferation, and survival (198, 199)
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People who carry the genetic mutations that cause G6PD deficiency are at an increased risk for hemolytic anemia when taking certain medications, including quinine-based medication
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