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Amylin signaling is closely connected to nutrient handling, gastric motility, and postprandial metabolic responses, which makes cagrilintide relevant in experimental models examining glycemic control and endocrine-metabolic communication [1]

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It increased survival of cultured enteric neurons and proliferation of enteric glial cells, suggesting direct neuroprotective effects on the enteric nervous system that could influence central serotonin tone through vagal signaling

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