It functions by inhibiting bacterial cell wall synthesis, leading to bacterial cell death
doi: 10.1016/j.diabet.2020.06.005 164 LeeYHKimSHKangJMHeoJHKimDJParkSHet al
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was supported by NASA through the NASA Hubble Fellowship grant #HST-HF2-51570.001-A awarded by the Space Telescope Science Institute, which is operated by the Association of Universities for Research in Astronomy, Incorporated, under NASA contract NAS5-26555
Compared to BPC-157, which acts primarily via the nitric oxide and growth hormone receptor pathways, and TB-500, which functions through actin sequestration and VEGF signalling, GHK-Cu operates largely through copper-mediated gene expression modulation and extracellular matrix remodelling
