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Earlier attainment of a mechanically competent boneimplant interface plausibly shortens the period during which microbial challenge and mechanical perturbation are most likely to initiate a trajectory toward crestal bone instability and peri-implant inflammation

In response to low oxygen tension, HIF-1 activates the transcription of a broad array of pro-angiogenic genes, most notably vascular endothelial growth factor A (VEGF-A) (167)