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Oxidative stress-induced intestinal injury has been reduced by the addition of SOD, glutathione peroxidase, and N-acetylcysteine, which reduces intestinal tissue tumor necrosis factor-a concentrations with anti-inflammatory and antioxidant properties [69, 70]

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More and more research suggests that effective treatment of DCM requires a specific endogenous antioxidant defense system, rather than non-selective scavenging of ROS that may impair physiological redox signaling (Hu et al., 2018

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