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In parallel, AOD 9604 has been shown to modulate enzymes involved in lipid synthesis, including acetyl-CoA carboxylase

9, 45 Also, they appeared to be a benefit in patients who did not have evidence of pre-existing glucose intolerance as measured by glycosylated hemoglobin

After treatment, a shift of phospholipid species from phosphatidylcholine to lysophosphatidylcholine was observed for both disease groups, presumably caused by an increase in anti-inflammatory lipid mediators (lipoxins and resolvins)