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In sickle cell anemia oxidative stress can trigger eryptosis and the release of red blood cell microparticles (RBC-MPs), which are small phospholipid-containing vesicles implicated in physiological processes such as hemostasis and potentially linked to macrovascular dysfunction

doi: 10.1007/s13300-021-01113-y [DOI] [PMC free article] [PubMed] [Google Scholar] 124

The authors conclude that the indicated SOD2 and SOD3 polymorphisms may be genetic risk factors for the susceptibility and progression of vitiligo suggesting the increased activity of SOD isoforms caused by the variant gene polymorphisms may lead to the accumulation of H 2 O 2 in cytoplasmic, mitochondrial and extracellular compartments resulting in melanocyte oxidative damage

The data reported are alarming in consideration of the highest levels of mycotoxins detected in foods for infants, who are the most vulnerable consumers

It has low bioavailability because the tripeptide is the substrate of proteases (peptidases) of the alimentary canal, and due to the absence of a specific carrier of glutathione at the level of the cell membrane