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Here, we confirm previously reported evidence of redox abnormalities in asthma (3), but greatly expand on these findings to identify a specific intracellular mechanism regulating these levels

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GLP-1R-dependent signaling is essential for the CNTF-induced cell proliferation, evidenced by the increased expression of GLP-1 in the hypothalamus of mice and the lack of efficacy in GLP-1R knockout mice

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