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2.2 Neurotoxic mechanisms METH induces neurotoxicity via a combination of oxidative stress, mitochondrial dysfunction, excitotoxicity, and neuroinflammation

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SU11274 inhibition of c-MET/HGF signalling We have previously characterised and described the efficacy of the specific small molecule inhibitor of c-MET (Sattler et al, 2003

On day 0, blood samples were collected via anesthesia-induced cardiac puncture and stored for plasma glycerol analysis