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Analysis of covariance was performed to control for potential confounding factors, such as age, education, sex, age of onset, and duration of illness

[20] designed a dual-layered gene + metabolic inhibition strategy by integrating Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR)-CRISPR-associated protein 9 (Cas9)-mediated USP22 knockdown with ND630, a small-molecule ACC1 inhibitor, thereby simultaneously suppressing lipid synthesis at the transcriptional and enzymatic levels