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Excessive generation of ROS within ovarian, adipose, and hepatic tissues overpowers antioxidant defenses, leading to oxidative damage of lipids, proteins, and mitochondrial DNA ( Androgen excess exacerbates redox disequilibrium by promoting NADPH oxidase activity and mitochondrial ROS production, while simultaneously impairing follicular maturation and oocyte quality ( Figure 1 represents the involvement of redox imbalance and endocrine dysregulation in the ovary, leading to hyperandrogenism or PCOS

Khuyn co: Nhiu khch hng phn hi mua phi hng gi, hng ht hn s dng hoc b mp mo, co rch tem trn cc sn thng mi in t hoc ni bn hng khng r ngun gc xut x

Kt qu sau 72 tun, ngi tham gia t mc gim cn t 16-22,5% trng lng c th

Canonical sampling through velocity rescaling

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