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mTOR signaling controls the recruitment and activation of innate, acquired immune cells and fibroblast-like synoviocytes (FLSs) during RA, resulting in the production of numerous chemokines, pro-inflammatory cytokines, and cathepsin to degrade extracellular matrix and cartilage, further contributing to the early characteristics of synovitis [3, 54] (Fig

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[DOI] [PMC free article] [PubMed] [Google Scholar] Awad J, Stotz HU, Fekete A, Krischke M, Engert C, Havaux M, Berger S, Mueller MJ