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This dual, seemingly paradoxical role of CUL5 across different tumor types warrants further investigation

These include enhancing intracellular TH availability via modulation of transporters such as monocarboxylate transporter 8 (MCT8) and deiodinases (e.g., DIO1), as well as directly targeting ECM remodeling through upregulation of MMP activity, inhibition of HA synthesis, and blockade of integrin-mediated signaling pathways (119121)

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ATB 0,+ is able to transport carnitine with low affinity when OCTN2 activity is limited [173]

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