Elevated fecal calprotectin levels are present in patients with both infectious and inflammatory conditions, including inflammatory bowel disease (IBD)
Continuous liver damage due to the overproduction of ROS and inflammation by diverse endogenous and exogenous injuries, failure at the respiratory chain in mitochondria [10,11], drop-in antioxidant elements such as superoxide dismutase [12], catalase activity [13], or the elevation of lipid peroxidation [14], among other described mechanisms, working together, are involved in injuries, apoptosis, inflammation, and fibrosis [10]
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CPPs can derive from bacteria and viruses or synthesized in the laboratory [241, 242]
Whatever the benefits are of this individualized approach, it is impractical at the population level