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In those with a genetic predisposition to T2D, it is widely believed that elevated concentrations of circulatory long-chain fatty acids (LC-FFA) significantly contribute towards the demise of insulin-producing pancreatic -cells the fundamental feature of the development of T2D

Mechanistically, much of this happens beyond its function as an osmolyte because betaine is also a methyl donor , [21,26] leading to tremendous benefits for the mitochondria

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