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Studies suggest that by displacing FOXO4, the peptide releases p53 to translocate to the cytoplasm and mitochondria, where it activates Bax/Bak pore formation and caspase-mediated apoptosis

Current evidence does not support TB-500 as a primary peptide for gastrointestinal repair

Animal studies report effects on inflammatory markers in models of gastric ulcers, inflammatory bowel conditions, and various organ injuries

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community-derived only