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The fact that the oxidation state of the active site cysteine of Prx can be transferred to other proteins allows Prx to function as a sensor of H 2 O 2 (Rhee et al., 2012)
The mechanism involves the interaction of SAM with SAMTOR, an S-adenosylmethionine sensor that regulates the mTORC1 pathway (Gu et al., 2017)
The upregulation of GPX6 expression found in these two studies suggests that its upregulation may have some antioxidant effects
Animal studies have emphasized the efficiency of the glymphatic system in purging waste from the brain, with peak effectiveness during sleep [14], particularly at night [204], and reduced efficacy observed in animal models of hypertension [205]
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