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In a cisplatin-induced AKI model, SS-31 was found to reduce mitochondrial and intracellular ROS levels, inhibit the expression of downstream NLRP3 vesicles, and improve renal oxidative stress and apoptosis [37]

Neither peptide has been through human clinical trials as a therapeutic agent all the safety data is preclinical

This integrated approach is one of the reasons patients at Formation consistently report stronger and faster results compared to individuals self-administering peptides without clinical guidance

BPC-157 dosage for tendon recovery depends on the injury, route of use, cycle length, health history, and provider guidance