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A study highlighted S100A1s ability to restore calcium cycling in cardiomyocytes, improving contractility in heart failure models, and gene therapy using AAV-S100A1 showed reduced fibrosis and hypertrophy in rodents (Rostami et al., 2025)

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Pour rappel, ce phnomne est connu pour tre lorigine de nombreux dgts cellulaires et tre impliqu dans le vieillissement prmatur des cellules

Some may support the release of brain-derived factors that protect neurons