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One potential mechanism is thought to involve activation of the central nervous system, with the area postrema (AP) and nucleus of the solitary tract (NTS) and the intermediolateral nucleus (IML) of the spinal cord similarly activated by administration of GLP-1RA 29,30

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Facilitating bloodbrain barrier crossing of GLP-1R agonists is sought after for other neurodegenerative diseases [36, 48]

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