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Reduced NNMT in adipose tissue was associated with increased expression of polyamine flux genes and increased adipocyte energy expenditure connecting NNMT inhibition to "futile cycling" mechanisms that consume energy without productive metabolic output

To further verify that the decreased resistance in the hr-52 mutant is caused by the interference with AT4G23100 gene, we obtained the previously reported mutant pad2-1 with the Ser298Asn substitution (S298N) within AT4G23100 21 to assess its responses to B

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