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The interplay between HAS3-mediated HA synthesis and HYAL1-driven degradation further exemplifies intrinsic temporal control of ECM remodeling, providing a natural biological model that parallels HAGSH scaffold responsiveness [19]

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This interaction leads to mRNA degradation or repression, forming a sophisticated regulatory network that is vital for proper cellular function and development, and is often implicated in various diseases (Figure 3) (Tabnak et al., 2021b

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