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To address these issues, most studies focus on the depletion of endogenous GSH by mediators such as nitric oxide (NO) 12 or Cu 2+
PMID 8697641
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Beyond CXCL16, additional mechanisms such as low-level CD36 expression, macropinocytotic uptake, and efferocytotic processes may facilitate the clearance of oxidized LDL (114)
When it occurs de novo, there is usually an intraepidermal junctional component
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