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The peptide also activates alternative angiogenic pathways including VEGF-independent routes via Src-caveolin-1-eNOS signaling, providing redundant mechanisms for vascular regeneration

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Notably, while initially demonstrated in patients with T2DM, recent landmark trials have shown that semaglutide also significantly reduces primary cardiovascular endpoint events (including cardiovascular causes, nonfatal myocardial infarction, or nonfatal stroke) in obese patients without diabetes, substantially expanding their potential cardioprotective applications