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SELENOP reduces tau aggregation by modulating tau protein phosphorylation and promotes the disaggregation of pre-formed tau tangles [111]

Thus, it might be a cytoprotection phenomenon with application in distinctive injuries and circumstances (the normalization of increased intraocular pressure, recovery of glaucoma along with the recovery of pupil function, retinal ischemia, and corneal sensitivity and lesion and maintained corneal transparency) [11,12,13,14,15,16,17]

It causes cell cycle arrest in the G2/M phase, leading to the downregulation of key proteins cyclin B1 and ccdc2 in the G2/M phase, while also enhancing the inhibition of oncogenes p21, Cip1, and p-p53

Importantly, the inflammatory environment may negatively affect the function of epithelial stem cells, and thus the reconstitution of healthy epithelium may be hindered [126,127,128,129]

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