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Beyond its classical metabolic role, research has found that DHODH can limit lipid peroxidation within mitochondria by maintaining its own mitochondrial CoQ/CoQH 2 redox pool, thereby independently suppressing ferroptosis ( 2 or ubiquinol, which acts as a lipophilic radical-trapping antioxidant that can effectively interrupt the lipid peroxidation chain reaction, thus establishing a defense line on the plasma membrane independent of the GPX4GSH system (Figure 1)

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In contrast, for wellness use with normal labs, the rationale is weaker and alternatives should be discussed

Acta 478 , 6267 (2018)