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Moreover, GLP-1 has a very rapid metabolism, with a half-life of 12 min in the plasma of mammals, and is rapidly inactivated mainly by the ubiquitous dipeptidyl peptidase-4 (DDP-4) enzyme and to a lesser degree by other peptidases [13]
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The metabolic burden of methyl donor deficiency with focus on the betaine homocysteine methyltransferase pathway
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