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When tissue folate and cobalamin stores are intact, an abnormal increment of postmethionine-load homocysteinemia may also reveal a saturation of the transsulfuration pathway caused by dietary methionine excess that might be combined with transsulfuration defects (in cases with basal hyperhomocysteinemia) or with the homozygous thermolabile MTHFR defect (in cases with normal fasting homocysteine) (Cattaneo, 1999)
The core pathology of type 2 diabetes is insulin resistance and beta cell failure, which leads to imbalance of energy distribution: reduced glucose uptake by muscle and adipose tissue, increased gluconeogenesis in liver, and increased glucose and free fatty acid (FFA) levels in blood
However, Mullins et al
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