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Mitophagy is impaired in HF, causing the accumulation of damaged mitochondria, due in part to reduced PGC-1 expression, and overexpression of PGC-1 restores mitochondrial function and cardiac performance in HF models [18]

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gingivalis , LPS, triggers extensive immune-inflammatory responses by binding to Toll-like receptor 4 (TLR4) on host cells, leading to tissue destruction and inducing persistent, low-grade systemic inflammation

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