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Archived from the original on October 30, 2013

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These agents cause sinoatrial (SA) and atrioventricular (AV) nodal depression and a decrease in myocardial contractility (nondihydropyridines)

In PCOS, the convergence of hyperandrogenismdriven adipokine dysregulation, insulin resistance, and macrophage infiltration arguably sets up a permissive environment for neutrophil activation: elevated adipokines such as leptin, resistin, and visfatin have been documented in PCOS and linked to chronic inflammatory signaling in adipose and reproductive tissues [90]