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Cardiac fibroblasts regulate myocardial proliferation through beta1 integrin signaling

Mechanistically, high-glucose-induced AGEs promote cuproptosis in cardiomyocytes by upregulating SLC31A1 expression, which drives pathogenic Cu accumulation [80]

Similar content being viewed by others Acknowledgements This work was supported by the Basic Science Research Program through the National Research Foundation of Korea (NRF) funded by the Ministry of Education (2020R1I1A3A04038150), Korea Basic Science Institute (National research Facilities and Equipment Center) grant funded by the Ministry of Education (2023R1A6C101A045), and the NRF grant funded by the Korea government (MSIT 2020R1A2C2103067)