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they target, bind to, and degrade disease-associated mRNAs, reducing the levels of proteins implicated in specific pathological conditions 6

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Regarding oxidative stress associated with mitochondrial complex I dysfunction and its role in BD, the importance of glutathione, an endogenous antioxidant, in preventing oxidative damage has been highlighted

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Regulation of TAM polarization TAMs mainly stem from monocytes originating in the bone marrow and are drawn to cancerous areas via the chemoattractant CCL2, which is produced by tumor cells [57]