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A plausible explanation of this apparent dichotomy is that p53 promotes cell survival by preventing excessive increases in ROS under moderate oxidative stress, whereas when the oxygen species increase over a threshold level, it switches to becoming a ROS inducer, triggering cell death

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Simultaneously, these agents suppress glucagon secretion from pancreatic alpha cells, further helping to regulate blood glucose levels

With advancements in materials science, targeted delivery of iron ions to bacteria to enhance oxidative stress enables specific bacterial elimination, while adjusting material components can protect host cells from ferroptosis [98, 99]

Role of physical and chemical interactions in the antibacterial behavior of ZnO nanoparticles against E