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The table below represents a clinical summary of the two therapies across key categories: Primary mechanism: GHK-Cu acts as a tissue repair signal

Pancreatic capillary blood flow in an improved model of necrotizing pancreatitis in the rat

For very high disease activity, you may even go right to an IL-17 blocker and bypass anti-TNFs, which have not proven as effective for ankylosing spondylitis as they have been for other types of inflammatory arthritis

This is why BPC-157 research expanded from tissue healing into psychiatry and neurogastroenterology

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