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In agreement with this, our results have showed a complete inactivation of AX by L-cysteine, but this involves a chemical reaction with amino groups rather than with the thiol groups of cysteine

Bulger EM, Maier RV (2001) Antioxidants in critical illness

These results indicate that VPA exposure induces significant oxidative stress by suppressing key antioxidant defense systems, and that hCiPSC-NSCs treatment effectively restores redox homeostasis through comprehensive enhancement of antioxidant capacity

In AD models, it improved memory even when given late, presumably by enhancing histone acetylation and BDNF expression [262]