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doi: 10.2147/JIR.S84993 42 ZhaoYYChenLHHuangLLiYZYangCZhuYet al

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The inductions of grxA and trxC are strong, but these genes do not play any obvious role under the H 2 O 2 stress conditions that we employed Under the conditions of our experiments, the level of H 2 O 2 inside the catalase/peroxide mutants rises to 12 micromolar, and the resultant activation of OxyR induces grxA 20-fold and trxC 30-fold (Figure 6A)

This process is governed by BCL2 family proteins, including the pro-apoptotic BCL2 associated X, apoptosis regulator (BAX) or BCL2 antagonist/killer 1 (BAK1, also known as BAK) as well as anti-apoptotic members, such as BCL2 apoptosis regulator (BCL2) and BCL2 like 1 (BCL2L1, also known as BCL-XL)