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4 Specific role of mitochondrial energy metabolic reprogramming in AKI During AKI, metabolic reprogramming in TECs may temporarily compensate for deficits in energy supply, support cellular survival, and partially mitigate tissue damage, thereby facilitating early repair processes

It might sound minor, but the distinction between these forms is really important

Epithalon also appears to inhibit the activity of Caspase-3, a key enzyme that is considered to promote apoptosis (programmed cell death)

Refined pores Studies show that tretinoin can reduce the appearance of large pores due to the increased cell turnover

doi: 10.1136/emj.7.3.148