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Under caspase-8 inhibition, RIPK1/RIPK3 phosphorylation initiates necrosome formation and MLKL activation, triggering membrane breakdown and the release of pro-inflammatory damage-associated molecular patterns (DAMPs) (Fritsch et al., 2019

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However, it is possible that in analogy to their role in neurological disorders, GAD65Ab are taken up by pancreatic beta cells and (a) uncouple the balanced regulation of insulin and glucagon secretion and (b) induce beta cell apoptosis through prolonged exposure to elevated extracellular glutamate levels

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doi:10.1016/j.rmed.2009.06.016