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These findings reveal that GGT1 safeguards against CaOx-induced nephrotoxicity by targeting GCLC to regulate ferroptosis and highlight GGT1 as a potential therapeutic target in nephrolithiasis

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Mitochondrial oxidative stress in heart failure: oxygen wastage revisited

When lacking or depletion of GSH, it may cause producing toxic effects or increasing the toxic effects of many chemicals or environmental factors