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doi: 10.1016/j.jaut.2022.102873 175 PatelAMLiuYSDaviesSPBrownRMKellyDAScheel-ToellnerDet al

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Liver metastatic breast cancer cells are recognized to depend on the HIF-1/PDK1 axis for their metabolic reprogramming to accelerate their efficient colonization and proliferation in the liver (138)

This initial phosphorylation cascade leads to autophosphorylation of the carboxy-terminal bidentate substrate-binding site (Tyr1349 and Tyr1356) of the c-Met and these residues have been shown as docking sites for downstream signaling molecules [52]