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The antiproliferative effect of calcitriol involves multiple pathways, such as the expression of cyclin-dependent kinase inhibitors (i.e., CDKN1A and CDKN1B), the production of growth factors (i.e., TGF-, IGF) and miRNAs, the regulation of cancer stem cell proliferation, tumor angiogenesis, epithelial-mesenchymal transition, and the activation of caspases (49, 5356)

Because it stays active in circulation far longer, it can be administered subcutaneously (under the skin rather than intramuscularly) and still reach muscle tissue systemically

These properties contribute to its favorable oral absorption, metabolic stability sufficient for once-daily dosing, and tissue distribution compatible with reaching GLP-1 receptors in the pancreas, gastrointestinal tract, and central nervous system

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