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In MDD, this peripheral-central coupling is likely amplified by BBB dysfunction characterized by increased permeability, neurovascular inflammation, and associations with symptom severity [172, 173] thereby lowering the threshold at which peripheral KP activation translates into central glutamatergic, oxidative, and neuroimmune dysregulation

Read next Hilary Brueck is a Health Correspondent at Business Insider, where she covers longevity and the quest to improve human aging

tissue microarray-based immunohistochemical study

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In two breast cancer studies, GLP1R agonism suppressed the production of pro-inflammatory cytokines (IL-8, VEGF) and promoted accumulation of CD8+ cytotoxic T-cells within the primary tumor, resulting in decreased proliferation and delayed tumor growth (76, 77)