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Furthermore, for individuals with MTHFR mutations or compromised liver function, methylcobalamin bypasses the metabolic bottlenecks that prevent the synthetic form from being utilized, ensuring the cells actually receive the active nutrient

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Further details on trial design development in ALS are referenced [53, 86,87,88,89]

This could improve overall fitness, increasing the likelihood of eligibility for liver transplantation and thereby potentially curing patients of their MASH-HCC

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