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Introduction The introduction of contemporary therapies for childhood T-cell acute lymphoblastic leukemia (T-ALL) has resulted in remission rates that are closer to that of B-cell precursor (BCP) ALL but survival rates remain lower and 15-20% of children with T-ALL die from relapsed/refractory disease.1 Patients with high-risk disease or those who relapse often receive more intensive treatment, making them more susceptible to toxicity and long-term secondary complications.2 This highlights the need to investigate other agents to treat this disease

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