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reported that upregulated transsulfuration pathway for cysteine synthesis in erastin-resistant ovarian cancer cells compensates for cysteine deprivation by xCT blockage, which is mediated by Nrf2-mediated CBS activation (51)

4 Gut microbiota dysbiosis and abnormal activation of the innate immune system 4.1 Evidence for the association between complement components and schizophrenia Current studies have identified elevated transcript levels of complement components (C1qA, C3, C4, C5) in patients with schizophrenia (114) (see Table 2 ), suggesting, albeit not entirely consistently, that increased activation of the classical complement pathway may be associated with SZ

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