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In addition, hypercalcemia, hyperphosphatemia, and hyperphosphateuria may occur

This pro-inflammatory state is further exacerbated by the reduced expression of PPAR and PPAR in zinc-deficient endothelial cells, as these receptors are crucial inhibitors of NF-B signaling, underlining zinc's vital role in preventing sustained inflammation [19]

Notably, this upregulation trend was particularly pronounced in BBIRE-T248 and PUC30 cell lines, which harbor only mutations in the Kras gene (Table 1) and show the greatest difference in terms of sensitivity to RSL3 between 2D and 3D (Fig

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