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The proposed mechanisms underlying these protective effects include the inhibition of several signaling pathways implicated in cancer initiation and progression, such as the phosphoinositide 3 kinase (PI3K)/protein kinase B (AKT)/mammalian target of rapamycin (mTOR), extracellular signal-regulated kinase/microtubule-associated protein kinase, and nuclear factor kappa-light-chain-enhancer of activated B cell pathways [32-34]

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