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Mitochondrial metabolic heterogeneity in CRC: implications for therapeutic divergence and precision intervention Building on earlier discussions of how mitochondrial metabolism contributes to resistance in CRC, driven by metabolic enzymes, intratumoural heterogeneity, and post-transcriptional regulatory networks, this section highlights the clinical relevance of metabolic divergence

The ferroptosis inhibitor Liproxstatin-1 (Lip-1) alleviated EAE development, improved motor function in mice, and prevented oligodendrocyte demyelination and neuronal death