Adams J, Baral M, Geis E, Mitchell J, Ingram J, Hensley A, Zappia I, Newmark S et al (2009) The severity of autism is associated with toxic metal body burden and red blood cell glutathione levels
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In parallel, the higher but transient P53 expression in Gstp1/2 / mice could contribute to the maintenance of the redox homeostasis and cell survival as well as the early increase in GSTM1 protein levels that blocks the TRAF2/ASK1 signaling pathway
These findings together imply that upregulation of Prxs renders motor neurons less susceptible to neurodegeneration, whereas breakdown of this redox system at late disease stages induces neuronal degeneration and accelerates disease progression (Kato et al., 2005)
