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Researchers are also investigating peptide-mediated pathways related to inflammation resolution, antioxidant defense mechanisms, and cellular repair processes within controlled research settings

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APS improve ER and the resulting autophagy by inhibiting the ATF6 passage and glucose-regulated protein 78 (GRP78)-related pathways [including IRE-1, the c-Jun N-terminal kinase (JNK), and PERK], reducing the apoptosis factors expression of C/EBP-homologous protein and caspase-12 (Wang et al., 2009

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